Type 2 diabetes is a very complicated topic, and I'm not sure if I'll ever get done talking about it on this site, there's quite a bit of info up here already and there's lots more to discuss as well.
I wanted to dedicate a post to looking to explain it in a simple way, for instance if I were giving a fairly brief talk to a group of people who only had a bit of knowledge about the disease and were also entrenched in a lot of the popular myths surrounding it, like for instance speaking to a group of medical doctors.
It is amusing and sad at the same time that people put their complete faith in doctors when it comes to treating their diabetes, folks who have very little knowledge or training in the field and basically just dispense medications according to their training. Of course their trainers are in the business of selling medications so that only makes sense really.
I always like to start on common ground, and the simple concept of type 2 diabetes being a disease of insulin resistance is something we can all agree on pretty much. You also hear that it's a deficiency of insulin so that's actually a good place to start.
In some cases type 2 does lead to that, and this is actually type 1. So some people have both, and we can see this in cases of the so called type 1.5, which is a combination of type 1 and type 2, or in some very late stage type 2's where their condition deteriorates to the point where they also become type 1, double diabetes.
There are also some type 1's that develop double diabetes, and this is actually more common, and this is because their insulin levels have been kept persistently high for a very long time, and they develop insulin resistance as type 2's do.
Insulin resistance equals insulin excess though, every time. This is what causes insulin resistance, too much insulin, and although we clearly know that hormonal resistance is caused by excess hormones, a lot of people close their eyes to this fact.
So we have 2 diseases here, type 1 is not enough endogenous insulin, type 2 is too much endogenous insulin, and a relatively small percentage of patients have both, not making enough insulin on their own and requiring insulin therapy to normalize levels, and being insulin resistant as well.
None of this so far should be in any sense controversial. Diabetes, any variety, is a hormonal disease. It is not a disease of high blood sugar, even though that is an easily observed symptom. When you get an infection, the fever or the inflammation you can get is not the disease, it's not a matter of getting rid of the fever or inflammation and then curing or even treating the disease.
This is the big mistake that conventional approaches to diabetes makes. The symptom, the high blood sugar, becomes the focus, and not even that big of a focus actually, as we tend to neglect things like the way our diets influence blood sugar and eat things we can't handle and look to reduce blood sugar with medication.
The high blood sugar is not the disease though. The disease of type 2 diabetes is hormonal imbalance, and while these imbalances are fairly complicated, the primary defect is insulin resistance caused by insulin excess.
So if you have an excess of a hormone and then seek to raise it, that just spells trouble, as you are making the disease worse over time. Sure, you can lower blood sugar in the short term, just like you can lower a fever with medications, but when you do this, the infection itself gets worse, and the infection is the disease.
So given that higher levels of insulin over time lead to greater insulin resistance, when we look to treat blood sugar by raising insulin, we make the insulin resistance, the disease, worse over time, and this is exactly what happens with conventional treatment of type 2.
If our insulin levels are abnormally high, and especially if we have diabetes as a result, the only sensible solution would be to look to lower insulin, normalize it, not raise it.
In the presence of elevated insulin levels, we actually have three choices here, to look to normalize it, to look to increase it and make the condition worse, or to do nothing. Doing nothing is actually the second best option, and the worst option, without a doubt, is to look to increase this excess further.
The state of one's insulin levels is not something to be assumed or guessed at, especially if one is completely confused about what these levels are generally at with the presentation of type 2 diabetes. So they assume it's too low because our blood sugar is too high, or they don't care what our levels are, they want to jack them up, blindly.
Diagnosis is supposed to be the first step in medical care though, and not diagnosing properly is reckless actually. Medical doctors use diagnostic tests to extreme, but with diabetes, they just measure your blood sugar and then just guess and guess wrongly in almost all cases.
It's not even that easy to get your insulin tested, because your doctor will most often tell you that it has no clinical value. This means that regardless of whether it is low, normal, or high, they want to raise it. This violates all principles of endocrinology as well as common sense, but no matter.
Some believe that if insulin resistance is present, then we require higher levels of insulin to normalize blood sugar, and that's OK and even preferable. That's just foolish though because once again this just makes insulin resistance worse, but moreover, it is the excess insulin itself that is behind type 2 diabetes.
The problem with type 2 diabetes isn't really high blood sugar, it's too much glucose and fat being put into cells. This is where another false assumption comes into play, an even bigger one, that assumes we have high blood sugar and this is because our cells are deficient in these nutrients.
The opposite is true though, our cells are being poisoned by too much glucose and fat already, that's what high insulin levels cause, and if you raise insulin levels, you make this worse. We know this for certain actually, it is not a guess, like their bad guesses.
So what is type 2 diabetes in a nutshell? Cellular glucotoxicity and lipotoxicity caused by excessive insulin levels. These toxicities are the real cause of high blood sugar, due to the dysregulation of glucose that they cause, where our bodies think that they are starving all the time and will dump massive amounts of extra glucose into our blood even though our blood sugar is already too high.
To fix this, or to even try, we need to address the real problem here, insulin toxicity. So we need to be diagnosing and treating our disease of high insulin, not seeking to make it worse by raising insulin to even more toxic levels.