Dr. Hite has a short video on his FAQ page which speaks about abdominal fat and what he believes are the precursors to diabetes, which he calls insulin fatigue. This is a model that’s widely believed to be the case and is actually based upon another big myth.
It is true that abdominal fat, visceral fat in particular, which is fat that accumulates around our abdominal organs, is the worst kind as far as the ill effects that fat causes. Fat around the pancreas in particular is bad, but not the fat itself, this is more like a canary in a coal mine, and where there is organ fat, there is lipotoxicity in the cells of the pancreas itself.
So it’s this internal fat that does us in, by poisoning both the beta and alpha cells. It’s the alpha cell poisoning that matters though as far as type 2 diabetes goes, the beta cells also get poisoned but this is not where the defect lies with diabetes.
Dr. Hite believes that type 2 diabetes occurs when the beta cells cannot keep secreting enough insulin to overcome insulin resistance. That’s actually false, it actually happens when the alpha cells secrete so much glucagon that even toxic amounts of insulin from the beta cells can no longer keep blood sugar down properly.
He speaks of the pancreas secreting higher than normal amounts of insulin during the period leading up to diabetes, but we know that this continues on long after one becomes diabetic, and high insulin levels, harmful ones actually, are a hallmark of type 2 diabetes. We know this to be the case because when we test type 2’s we see insulin levels several times higher than normal typically.
Advanced cases may exhibit more normal levels but glucagon remains high, and this disease is really all about glucagon levels as we’ve been discussing, so while this does take the load off so to speak, and glucagon may actually go down during this late stage, that alone doesn’t help because the insulin to glucagon ratios remain high.
The idea that our beta cells get fatigued or wear or or however you want to describe their downregulated state over time is not the problem here, and in fact they still secrete too much insulin, levels that poison the body, at this time. This is why it’s so important to reduce insulin levels, try to normalize them, as best you can, through diet and medication, and although this in itself may not fix things, it does go a long way toward achieving this, and is a necessary step actually.
This is discussed by Dr. Hite in looking to answer the question of whether or not a type 2 diabetic will require insulin, but the only sensible answer to this is, only if one actually becomes insulin deficient, which is possible but not typical at all.
In order to tell this though, insulin levels need to be monitored, and when a deficiency is discovered, we must dose to balance the hormone, not bash blood sugar like bumbling idiots. The latter is the choice that’s almost always taken, in spite of the sheer madness involved.
Since we know, or we should know if we even bother to look into all this, that high insulin levels cause several metabolic disorders including type 2 diabetes, and that our biggest problem is insulin resistance, and they even admit that, and insulin resistance is caused by too much insulin, something we know with certainty, why in the world would we elevate insulin further when it is already too high and making us sick?
Guess what happens when we do this, we get sicker. How in the world would anyone even contemplate doing this?
Well this comes down to what amounts to stupidity actually. They look at correlations between blood sugar and microvascular damage and they see that, not surprisingly, lower blood sugar correlates with less risk of this damage, and vice versa. These are long term correlations though, for instance an A1C of 7 controls these risks pretty well, and this has become the target of the ADA, 7 or under. 8 is riskier, 9 even riskier, and so on.
So increasing insulin further does lower blood sugar temporarily, although none of the protocols for doing this provides long term benefits, just short ones. When we look down the road, and that’s what we’re out for, long term control here, these treatments fail, because they worsen insulin resistance and in spite of increasing the medication over time, blood sugar goes up from that anyway.
So later on you’ve worsened insulin resistance by a lot, people need ridiculously high levels of it just to avoid truly bad blood sugar, this also makes you fat and dramatically increases your risk of macrovascular complications like heart attacks and strokes, because that’s just what toxic levels of insulin does.
Sadly though, this fails at even what it is out to treat, high blood sugar, you end up with high blood sugar, very high insulin now because you need that much to keep it from going even higher, you’re so insulin resistant, and you may die of a heart attack as well now.
This is what happens when you worsen hormonal imbalances on purpose, a preposterous idea actually. So the real answer to this question is, one may have to inject oneself with insulin but that should only be considered if it is needed to restore insulin levels to normal, but there’s absolutely no reason that this should ever happen to a type 2 diabetic, with the proper care that is.
The proper care starts with addressing the real problem of type 2 diabetes, insulin resistance caused by too much insulin, and you won’t fix that by injecting insulin or taking pills to make your pancreas go even crazier, although this can lower blood sugar for a time, the long term affects of such a protocol are deplorable.
So when he speaks of loss of function of the pancreas, he is speaking about our beta cells downregulating their insulin production to try to save us from all this, and if not for this diabetes would progress even faster than it does. The last thing we need when our insulin levels are already too high is more insulin, we’re getting poisoned enough from it already without making this all worse.