Diabetes Basics Part 13

type 2 diabetics

We left off Part 12 talking a little about the various types of diabetes, and while this doesn’t require too much discussion, it’s good to know what you have, what the disease actually is, and it ain’t high blood sugar by the way, that’s just one of the symptoms.

Dr. Jason Fung describes our approach to treating diabetes uses an analogy of looking at someone with a fever and then trying to fight the fever because this is the outward manifestation of what appears to be wrong.  This is an excellent analogy, and by the way Dr. Fung has some excellent videos on You Tube for those interested in learning more about the disease.  He’s one of a select number of medical doctors who has opened his mind to the causes and proper treatment of diabetes by the way and his videos are simple enough for anyone to follow along with, and his observations are backed with plenty of science.

So what happens if we treat an infection by just looking to reduce the fever that comes with it?  Well the fever just gets worse, because the fever not only is just a symptom of the condition, it’s actually one of the ways the body uses to fight it off, by raising our temperature so that the offending bacteria can be more easily controlled, as they don’t like higher temperatures.

So if all we are looking to do is lower our temperature, we aren’t helping ourselves.  We need to get at what is behind this, the bacterial infection in this case, and of course we do because we’re not that stupid.

With treating diabetes though, we do tend to be that stupid, as we take one symptom of what is wrong with us, high blood sugar, and just focus on that, and ignore what may be behind it, we’re not really even interested in treating what is behind it.

We say, well type 2 it’s either insulin deficiency or insulin resistance, and we speak of these things like past events that have perhaps led up to the diagnosis of the condition, but once we’re diagnosed, we just suspend our thinking and go to work on the high blood sugar, the symptom, pretty much exclusively.

So there’s the claim that we are sometimes insulin deficient, that’s actually type 1 diabetes, but how would they know as they aren’t interested in even measuring how much insulin we are producing, and if they were, they would learn that we make too much, not too little.  Since they are only interested in focusing on blood sugar, insulin levels only matter if they suspect autoimmune diabetes, type 1, the one that comes with deadly low levels of insulin, and if they suspect that, then they will test our insulin, but they are not at all interested in the insulin levels of type 2’s.

With the other thing behind type 2, insulin resistance, well it’s taken for granted that we’re insulin resistant, no one wants to measure how resistant we are or how this might be changing over time, you have to measure both blood sugar and insulin levels for that, they don’t care though.

They might say, well we do have a class of meds called TZDs which overpower peripheral insulin resistance, so we do have a way to treat it, but peripheral insulin resistance isn’t the problem and its actually part of the body’s way of protecting itself from all that glucose in the blood, protection from excesses of both glucose and fat actually.

So TZDs overpower this, people get fat, people get sicker, people die as well.  These drugs have been taken off the market in Europe because they just kill too many people, although in the United States, where 100,000 people die a year from taking prescription drugs as directed, they have a higher tolerance for drug induced death.

So as a compromise, this stuff can only be prescribed as a last resort, when all else fails, although these rules are not always strictly followed, and doctors basically do what they want.

So if they just reduce insulin resistance so to speak, and insulin resistance is our problem, why all the trouble?  Because peripheral insulin resistance is not our problem, it is insulin resistance in the pancreas and liver that does us in.  Increasing insulin to overcome that, or to try to and do so temporarily, is bad enough, but the body at least has a way to protect itself from too much damage being done, and that’s by just increasing the peripheral insulin resistance.

It does not have such a natural defense against TZDs though, which are unique substances not found in the body nor in nature, and this has its way with the cells even more, they get poisoned even more, people get fat from the stuff even more because we’re now storing even more toxic levels of nutrients, things just end up uglier.

The funny thing is, and this really would be funny if the story wasn’t such a sad one, is that all they care about is how much glucose we have in the blood.  They don’t care about how much fat is in our blood, as free fatty acids, which by the way is increased by high levels of insulin, and it’s these free fatty acids that do the most damage to us.

They don’t care if we infuse all this extra stuff into cells, the more the better, let’s just work on the outward manifestation of the disease, how much sugar is in the blood, and let’s not worry what happens when we just cram all that glucose and all that fat into cells, as much as we can, much like someone would clean their room by stuffing everything under the bed, with toxic mold growing there, and when we smell it and react to it all we do is keep stuffing crap under there.

When you do this with insulin by the way, you not only have all this extra cramming, but insulin works beautifully in also getting too much of this stuff into our blood in the first place, too much fat from converting too much carbohydrate into fat, and too much glucose from this fat poisoning our pancreas.

So we need to stop worrying about the fever here and deal with the infection, and deal with it in a way that is effective in seeking to make us well, but the conventional approach to diabetes misses the boat here completely.

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